Search results for " 5-HT3"

showing 10 items of 10 documents

Single-dose Palonosetron for prevention of chemotherapy-induced nausea and vomiting in patients with aggressive non-Hodgkin's lymphoma receiving mode…

2011

PURPOSE: The control of nausea and vomiting induced by chemotherapy is paramount for overall treatment success in cancer patients. Antiemetic therapy during chemotherapy in lymphoma patients generally consists of anti-serotoninergic drugs and dexamethasone. The aim of this trial was to evaluate the efficacy of a single dose of palonosetron, a second-generation serotonin type 3 (5-HT(3)) receptor antagonist, in patients with aggressive non-Hodgkin's lymphoma receiving moderately emetogenic chemotherapy (MEC) containing steroids. METHODS: Patients received a single intravenous bolus of palonosetron (0.25 mg) before administration of chemotherapy. Complete response (CR) defined as no vomiting …

MaleQuinuclidinesmedicine.medical_treatmentCINVCHOPGastroenterologyDexamethasoneSettore MED/15 - Malattie Del SangueAntineoplastic Combined Chemotherapy ProtocolsSerotonin 5-HT3 Receptor AntagonistsProspective StudiesAged 80 and overLymphoma Non-HodgkinPalonosetronNauseaMiddle AgedEmesisPalonosetronTreatment OutcomeOncologyAnesthesiaCHOP CINV Emesis Nausea NHL PalonosetronVomitingFemaleOriginal Articlemedicine.symptomCHOP-CINV; emesis; nausea; NHL; Palonosetronmedicine.drugAdultmedicine.medical_specialtyVomitingNauseamedicine.drug_classNHLYoung AdultInternal medicinemedicineHumansAntiemeticGlucocorticoidsAgedChemotherapybusiness.industryIsoquinolinesmedicine.diseaseNon-Hodgkin's lymphomaAntiemeticsbusinessCHOPChemotherapy-induced nausea and vomiting
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Antidepressants and Antipsychotic Drugs Colocalize with 5-HT(3) Receptors in Raft-Like Domains

2005

Despite different chemical structure and pharmacodynamic signaling pathways, a variety of antidepressants and antipsychotics inhibit ion fluxes through 5-HT3receptors in a noncompetitive manner with the exception of the known competitive antagonists mirtazapine and clozapine. To further investigate the mechanisms underlying the noncompetitive inhibition of the serotonin-evoked cation current, we quantified the concentrations of different types of antidepressants and antipsychotics in fractions of sucrose flotation gradients isolated from HEK293 (human embryonic kidney 293) cells stably transfected with the 5-HT3Areceptor and of N1E-115 neuroblastoma cells in relation to the localization of …

Fluphenazinemedicine.medical_specialtyPharmacology5-HT3 receptorCell LineMembrane MicrodomainsDesipramineInternal medicinemedicineHumansSerotonin 5-HT3 Receptor AntagonistsReceptorClozapine5-HT receptorbiologyChemistryGeneral NeuroscienceAntidepressive AgentsEndocrinologybiology.proteinSerotoninSerotonin AntagonistsSignal transductionReceptors Serotonin 5-HT3medicine.drugCellular/MolecularAntipsychotic AgentsProtein Binding
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Involvement of cholinergic nicotinic receptors in the menthol-induced gastric relaxation.

2014

We have previously demonstrated that menthol reduces murine gastric tone in part through a neural mechanism, involving adrenergic pathways and reduction of ongoing release of acetylcholine from enteric nerves. In the present study we aimed to verify whether the gastric relaxation to menthol may be triggered by interaction with neural receptors or ionic channels proteins, such as transient receptor potential (TRP)-melastatin8 (TRPM8), TRP-ankyrin 1 (TRPA1), 5-hydroxytriptamine 3 (5-HT3) receptor or cholinergic nicotinic receptors. Spontaneous mechanical activity was detected in vitro as changes in intraluminal pressure from isolated mouse stomach. Menthol (0.3-30 mM) induced gastric relaxati…

AgonistMalemedicine.medical_specialtySerotoninmedicine.drug_classDimethylphenylpiperaziniumMuscle RelaxationTRPM Cation ChannelsPharmacologyReceptors NicotinicSettore BIO/09 - Fisiologiachemistry.chemical_compoundMiceGanglion type nicotinic receptorTransient Receptor Potential ChannelsIsothiocyanatesInternal medicinemedicineTRPM8AnimalsSerotonin 5-HT3 Receptor AntagonistsRNA MessengerTRPA1 Cation ChannelPharmacologyStomachmenthol gastric relaxation TRPM8 TRPA1 nicotinic receptorsReceptor antagonistOndansetronMice Inbred C57BLMentholEndocrinologyNicotinic agonistchemistryPurinesHexamethoniumAcetanilidesAlpha-4 beta-2 nicotinic receptorGastrointestinal Motilitymedicine.drugEuropean journal of pharmacology
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Antidepressants are functional antagonists at the serotonin type 3 (5-HT3) receptor

2003

Antidepressants are commonly supposed to enhance serotonergic and/or noradrenergic neurotransmission by inhibition of neurotransmitter reuptake through binding to the respective neurotransmitter transporters or through inhibition of the monoamine oxidase. Using the concentration-clamp technique and measurements of intracellular Ca2+, we demonstrate that different classes of antidepressants act as functional antagonists at the human 5-HT3A receptor stably expressed in HEK 293 cells and at endogenous 5-HT3 receptors of rat hippocampal neurons and N1E-115 neuroblastoma cells. The tricyclic antidepressants desipramine, imipramine, and trimipramine, the serotonin reuptake inhibitor fluoxetine, t…

Serotoninmedicine.medical_specialtySerotonin reuptake inhibitorMirtazapinePharmacologyKidneySerotonergicHippocampusNoradrenergic and specific serotonergic antidepressantCell LineMembrane PotentialsReuptakeNeuroblastomaCellular and Molecular NeuroscienceNorepinephrine reuptake inhibitorCell Line TumorInternal medicineDesipraminemedicineAnimalsHumansSerotonin 5-HT3 Receptor AntagonistsPharmacology (medical)Molecular BiologyNeuronsChemistryGeneral MedicineTrimipramineAntidepressive AgentsRatsPsychiatry and Mental healthEndocrinology5-HT6 receptorCalciumReceptors Serotonin 5-HT3medicine.drugPharmacopsychiatry
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Endotoxaemia resulting from decreased serotonin tranporter (5-HTT) function: A reciprocal risk factor for depression and insulin resistance?

2015

International audience; Depression and diabetes are serious diseases with an increasing global prevalence. Intriguingly, recent meta-analyses have highlighted an asymmetrical relationship between the two conditions as depressed patients were found to display a higher risk of developing type 2 diabetes than those individuals suffering from diabetes are to become depressed. Based on recent findings, we favor a hypothesis where by decreased peripheral serotonin (5-HT) transporter (5-HTT) function is a reciprocal risk factor for the comorbidity of depression and diabetes, as it can trigger inflammatory pathogenetic mechanisms of both conditions. Higher intestinal levels of 5-HT and 5-HT3 recept…

Serotoninmedicine.medical_specialty5-HTAntidepressantComorbidityType 2 diabetesBehavioral NeuroscienceInsulin resistanceDiabetes mellitusInternal medicineAnimal models of depressionmedicineAnimalsHumansDepression (differential diagnoses)Serotonin Plasma Membrane Transport ProteinsInflammationIntestinal permeabilitybiologybusiness.industryDepressionInsulin resistancemedicine.diseaseAntidepressive AgentsReceptor InsulinEndotoxemia3. Good healthInsulin receptorEndocrinologyDiabetes Mellitus Type 2Immunologybiology.proteinAntidepressant[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Serotonin 5-HT3businessSignal Transduction
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α-Conotoxins EpI and AuIB switch subtype selectivity and activity in native versus recombinant nicotinic acetylcholine receptors

2003

The Xenopus laevis oocyte expression system was used to determine the activities of alpha-conotoxins EpI and the ribbon isomer of AuIB, on defined nicotinic acetylcholine receptors (nAChRs). In contrast to previous findings on intracardiac ganglion neurones, alpha-EpI showed no significant activity on oocyte-expressed alpha3beta4 and alpha3beta2 nAChRs but blocked the alpha7 nAChR with an IC50 value of 30 nM. A similar IC50 value (103 nM) was obtained on the alpha7/5HT3 chimeric receptor stably expressed in mammalian cells. Ribbon AuIB maintained its selectivity on oocyte-expressed alpha3beta4 receptors but unlike in native cells, where it was 10-fold more potent than native alpha-AuIB, had…

α7 nicotinic acetylcholine receptorα-Conotoxin AuIBRecombinant Fusion ProteinsBiophysicsXenopusNicotinic AntagonistsReceptors NicotinicPharmacologyTransfectionBiochemistrycomplex mixturesSubstrate SpecificityInhibitory Concentration 50Xenopus laevisStructural BiologyGeneticsmedicineAnimalsConotoxinNicotinic AntagonistReceptorMolecular BiologyAcetylcholine receptorbiologyα-Conotoxin EpICell Biologybiology.organism_classificationRatsCell biologyProtein SubunitsNicotinic acetylcholine receptorNicotinic agonistnervous systemIntracardiac gangliaOocytessense organsReceptors Serotonin 5-HT3ConotoxinsAcetylcholineXenopus laevis oocytemedicine.drugFEBS Letters
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5-HT3 receptor-channels coupled with Na+ influx in human T cells: role in T cell activation.

1999

The study was conducted on a human (Jurkat) T cell line, loaded with a Na+ fluorescent probe, SBFI/AM. Serotonin and an agonist of 5-HT3 receptor-channels, 2-methyl-5HT, evoked Na+ influx, whereas the agonists of other serotonergic receptor subtypes, i.e., 5-HT1A and 5-HT1B receptors, failed to induce Na+ influx in these cells. By using 3H-BRL43694, an agonist of 5-HT3 receptor-channels, we characterized 5-HT3 lymphocyte receptors which exhibited a density (Bmax) of 300 +/- 20 fmol/10(6) cells and a Kd of 30 nM in Jurkat T cells. The T-cell 5-HT3 receptor-channel is not regulated either by the protein kinase C or by the free intracellular calcium concentrations as the agents known to activa…

AgonistSerotoninmedicine.drug_classMetoclopramideT cellT-LymphocytesImmunologyBiologyLymphocyte ActivationJurkat cellsCalcium in biologyPiperazinesSodium ChannelsGranisetronJurkat CellsQuinoxalinesmedicineImmunology and AllergyHumansCalcium SignalingPhytohemagglutininsReceptorProtein kinase C5-HT receptorProtein Kinase C8-Hydroxy-2-(di-n-propylamino)tetralinIon TransportRyanodineCell CycleSodiumCell biologyNeoplasm ProteinsSerotonin Receptor AgonistsEnzyme Activationmedicine.anatomical_structureNeurologyReceptors SerotoninReceptor Serotonin 5-HT1BThapsigarginNeurology (clinical)Serotonin AntagonistsReceptors Serotonin 5-HT3Ion Channel GatingReceptors Serotonin 5-HT1IntracellularJournal of neuroimmunology
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One shot NEPA plus dexamethasone to prevent multiple-day chemotherapy in sarcoma patients

2019

Purpose: Chemotherapy-induced nausea and vomiting (CINV) is one of the most feared and disturbing adverse events of cancer treatment associated with decreased adherence to effective chemotherapy regimens. For high-risk soft tissue sarcoma patients, receiving multiple-day chemotherapy (MD-CT), antiemetic guidelines recommend a combination of an NK 1 receptor antagonist (NK 1 -RA), a 5-HT 3 receptor antagonist (5HT 3 -RA), and dexamethasone on each day of the antineoplastic treatment. NEPA is the first oral fixed-dose combination of a highly selective NK 1 -RA, netupitant, and second-generation 5HT 3 -RA, palonosetron. So far, no data has been published in literature about the efficacy of a s…

MaleOncologyQuinuclidinesMultiple-dayPyridinesmedicine.medical_treatmentCINVPilot ProjectsDexamethasonechemistry.chemical_compound0302 clinical medicineNeurokinin-1 Receptor AntagonistsClinical endpointSerotonin 5-HT3 Receptor AntagonistsProspective Studies030212 general & internal medicineAged 80 and overSoft tissue sarcomaPalonosetronNauseaSarcomaMiddle AgedReceptors Neurokinin-1PalonosetronOncology030220 oncology & carcinogenesisVomitingFemaleOriginal Articlemedicine.symptommedicine.drugAdultmedicine.medical_specialtyVomitingmedicine.drug_classNauseaAntineoplastic Agents03 medical and health sciencesInternal medicinemedicineHumansAntiemeticNetupitantAuthor CorrectionAgedChemotherapybusiness.industryIsoquinolinesmedicine.diseasechemistryNetupitantAntiemeticsbusinessSupportive Care in Cancer
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Antipsychotic drugs antagonize human serotonin type 3 receptor currents in a noncompetitive manner

2004

The serotonin type 3 (5-HT(3)) receptor is the only ligand-gated ion channel receptor for serotonin (5-HT). 5-HT(3) receptors play an important role in modulating the inhibitory action of dopamine in mesocorticolimbic brain regions. Neuroleptic drugs are commonly thought to exert their psychopharmacological action mainly through dopamine and serotonin type 2 (5-HT(2)) receptors. Except for clozapine, a direct pharmacological interaction of neuroleptics with 5-HT(3) receptors has not yet been described. Using the concentration-clamp technique, we investigated the effects of flupentixol, various phenothiazines, haloperidol, clozapine and risperidone on Na(+)-inward currents through 5-HT(3) re…

medicine.medical_specialtyPharmacologyKidney5-HT3 receptorCell LineMembrane PotentialsMiceNeuroblastomaCellular and Molecular NeuroscienceDopamineCell Line TumorInternal medicinemedicineAnimalsHumansCalcium SignalingReceptorMolecular BiologyDose-Response Relationship DrugbiologyBrain NeoplasmsChemistryFlupentixolPsychiatry and Mental healthEndocrinologyDopamine receptorCompetitive antagonistbiology.proteinLigand-gated ion channelCalciumSerotoninReceptors Serotonin 5-HT3Ion Channel GatingAntipsychotic AgentsSignal Transductionmedicine.drugMolecular Psychiatry
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A Role for the β1-β2Loop in the Gating of 5-HT3Receptors

2005

Based on theTorpedoacetylcholine receptor structure, Unwin and colleagues (Miyazawa et al., 2003; Unwin, 2005) hypothesized that the transduction of agonist binding to channel gate opening involves a “pin-into-socket” interaction between αV46 at the tip of the extracellular β1-β2loop and the transmembrane M2 segment and M2-M3 loop. We mutated to cysteine the aligned positions in the 5-HT3Aand 5-HT3Bsubunit β1-β2loops K81 and Q70, respectively. The maximal 5-HT-activated currents in receptors containing 5-HT3A/K81C or 5-HT3B/Q70C were markedly reduced compared with wild type. Desensitization of wild-type currents involved fast and slow components. Mutant currents desensitized with only the f…

XenopusMolecular Sequence DataGatingCell Linelaw.inventionMicelawExtracellularAnimalsHumansAmino Acid SequenceReceptorIon channelAcetylcholine receptorChemistryGeneral NeuroscienceWild typeProtein Structure TertiaryRatsBiochemistryMutagenesis Site-DirectedBiophysicsFemaleReceptors Serotonin 5-HT3Ion Channel GatingTorpedoCellular/MolecularCysteineThe Journal of Neuroscience
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